This study mainly found evidence for the social causation theory in early adolescence, when lower education predicted increases in subsequent alcohol use. The study found no evidence in support of the health-related selection hypothesis with respect to alcohol use. By determining initial educational level, childhood characteristics also predict subsequent trajectories in alcohol use.

Author

Heiko Schmengler (email: h.schmengler@uu.nl), Margot Peeters, Anton E. Kunst, Albertine J. Oldehinkel, Wilma A. M. Vollebergh

Citation

Schmengler H, Peeters M, Kunst AE, Oldehinkel AJ, Vollebergh WAM (2022) Educational level and alcohol use in adolescence and early adulthood—The role of social causation and health-related selection—The TRAILS Study. PLoS ONE 17(1): e0261606. https://doi.org/10.1371/journal.pone.0261606


Source
PLoS ONE
Release date
19/01/2022

Educational Level and Alcohol Use in Adolescence and Early Adulthood—the Role of Social Causation and Health-Related Selection—the TRAILS Study

Abstract

Introduction

Both social causation and health-related selection may influence educational gradients in alcohol use in adolescence and young adulthood. The social causation theory implies that the social environment (e.g. at school) influences adolescents’ alcohol use behavior. Conversely, the health-related selection hypothesis posits that alcohol use (along other health-related characteristics) predicts lower educational attainment.

From past studies it is unclear which of these mechanisms predominates, as alcohol use may be both a cause and consequence of low educational attainment. Furthermore, educational gradients in alcohol use may reflect the impact of ‘third variables’ already present in childhood, such as parental socioeconomic status (SES), effortful control, and IQ.

Method

This study investigated social causation and health-related selection in the development of educational gradients in alcohol use from adolescence to young adulthood in a selective educational system. The researchers used data from a Dutch population-based cohort (TRAILS Study; n = 2,229), including measurements of educational level and alcohol use at ages around 14, 16, 19, 22, and 26 years (waves 2 to 6).

First, the researchers evaluated the directionality in longitudinal associations between education and alcohol use with cross-lagged panel models, with and without adjusting for pre-existing individual differences using fixed effects.

Second, the researchers assessed the role of childhood characteristics around age 11 (wave 1), i.e. IQ, effortful control, and parental SES, both as confounders in these associations, and as predictors of educational level and alcohol use around age 14 (wave 2).

Results

In fixed effects models, lower education around age 14 predicted increases in alcohol use around 16. From age 19 onward, the researchers found a tendency towards opposite associations, with higher education predicting increases in alcohol use. Alcohol use was not associated with subsequent changes in education. Childhood characteristics strongly predicted education around age 14 and, to a lesser extent, early alcohol use.

Conclusion

This study mainly found evidence for the social causation theory in early adolescence, when lower education predicted increases in subsequent alcohol use. The study found no evidence in support of the health-related selection hypothesis with respect to alcohol use. By determining initial educational level, childhood characteristics also predict subsequent trajectories in alcohol use.


Source Website: PLOS ONE