This study found that children with prenatal exposure to alcohol had deoxygenated hemoglobin (HBR) levels in the medial prefrontal cortex (PFC) which were negatively related to externalizing problems and levels in the medial and right lateral PFC were positively related to errors on a cognitive inhibition task.

Author

Julie A. Kable, Claire D. Coles, Sarah N. Mattson, and CIFASD

Citation

Kable JA, Coles CD, Mattson SN. Neurodevelopmental Outcomes Associated with Prefrontal Cortical Deoxygenation in Children with Fetal Alcohol Spectrum Disorders. Dev Neuropsychol. 2020 Jan-Feb;45(1):1-16. doi: 10.1080/87565641.2020.1712604. Epub 2020 Jan 8. PMID: 31914808; PMCID: PMC7080191.


Source
Developmental Neuropsychology
Release date
08/01/2020

Neurodevelopmental Outcomes Associated With Prefrontal Cortical Deoxygenation in Children With Fetal Alcohol Spectrum Disorders

Abstract

Relationships between neurodevelopmental functioning and hemodynamic changes in the prefrontal cortex (PFC) were contrasted between children with prenatal alcohol exposure (PAE) and children who differed relative to their history of PAE and the presence of other neurodevelopmental impairments.

For all groups, deoxygenated hemoglobin (HBR) levels in the medial PFC area were negatively related to externalizing problems and levels in the medial and right lateral PFC were positively related to errors on a cognitive inhibition task. Hemodynamic changes in the medial and right lateral PFC of children with PAE demonstrated stronger relationships to aspects of executive functioning relative to contrast groups.


Source Website: NCBI